Tryptophan usually is the precursor to serotonin so wouldn't generally affect dopamine production.

Tyrosine is the main AA for dopamine, but its conversion to L-DOPA is the rate-limiting step, so supplementing it doesn't really help. You need to give L-DOPA to bypass that reaction if you want to bump dopamine levels.

Yeah it's weird isn't it.

I personally can't imagine caring about more money past a certain amount, but I think the process of becoming that wealthy weeds out people like us who don't want it enough.

Thus, the very richest are always, by a process of elimination, people for whom no amount is sufficient.

Or that's my theory anyway.

There's no easy answer to this one.

It depends what kind of evidence you want to consider (ie human clinical data vs. animal models) and what you count as neuroinflammation (ie cytokine concentrations, gene expression patterns, peripheral immune cell infiltration).

Human clinical data is sparse, variable and usually reliant on sub-par proxy measures so it's difficult to say. Animal data will tell you they do, but you need to pay careful attention to exactly what each study did, what they measured and whether it's of any relevance to a human disease.

If you want to start reading more about it, review articles can help but they are often specific to a particular pathological setting. To go into the primary literature you really need to pick a specific scenario and read around that.

Sorry I couldn't be any more specific, but there really isn't a short or satisfying answer of any value.