Submitted by fourleafedrover8 t3_yji28e in askscience
I have a burning desire to know this information (am currently ill and cannot stop sneezing)!
Submitted by fourleafedrover8 t3_yji28e in askscience
I have a burning desire to know this information (am currently ill and cannot stop sneezing)!
the_original_Retro t1_iur0p2c wrote
It's kind of neither.
It's important to understand that the virus doesn't "want" anything. It's like a little emotionless and purposeless robot. It reproduces because its programming tells it to reproduce, and that's it. So it's all about that program and what that program tells the virus to do. And occasionally that program up and changes a little thanks to viral mutations.
So let's look at two newly mutated types of virus.
One virus type has a newly written section of program code that triggers the body to sneeze by causing the infected body to generate more mucus and irritate nasal membranes that create the necessary "tickle". In other words, its programming activates our body's natural triggers to have to sneeze. That sneeze will create an aerosol and scatter liquid droplets that contain virus particles. This causes the virus to spread around and cover more real estate.
The other virus type does not share this new programming code. It doesn't trigger sneezing. So, because there are no sneezes, it spreads less effectively.
The first virus's program is more effective at increasing the odds that it will be spread to other hosts... and so it survives and propagates more, while the other virus doesn't spread beyond its initial host as effectively, and propagates less. The first virus doesn't MEAN to do this, it's just the way its programming changed to allow it to spread. And so the virus ends up multiplying in more bodies without "intending" to do so.
It's the result of an accident that's lucky for the virus... but unlucky for us.